Visfatin regulates Pg LPS-induced proinflammatory/prodegradative effects in healthy and inflammatory periodontal cells partially via NF-κB pathway - ScienceDirect
Contribution of Porphyromonas gingivalis lipopolysaccharide to experimental periodontitis in relation to aging. - Abstract - Europe PMC
In Vivo Up-Regulation of Kinin B1 Receptors after Treatment with Porphyromonas gingivalis Lipopolysaccharide in Rat Paw | Journal of Pharmacology and Experimental Therapeutics
GI Bacteria Affects Rheumatoid Arthritis Disease Markers | Chondrex, Inc.
P.g.-infection and LPS-PG induce myofibroblastic activation of HSCs.... | Download Scientific Diagram
IJMS | Free Full-Text | P. gingivalis-LPS Induces Mitochondrial Dysfunction Mediated by Neuroinflammation through Oxidative Stress
LPS-PG | Lipopolysaccharide from Porphyromonas Gingivalis
Porphyromonas gingivalis lipopolysaccharides act exclusively through TLR4 with a resilience between mouse and human | Scientific Reports
Inflammatory signals in human oral keratinocytes triggered by P.... | Download Scientific Diagram
LPS-PG | Lipopolysaccharide from Porphyromonas Gingivalis
Chlorogenic Acid as a Positive Regulator in LPS-PG-Induced Inflammation via TLR4/MyD88-Mediated NF-κB and PI3K/MAPK Signaling Cascades in Human Gingival Fibroblasts
Lipoxin A4 promotes autophagy and inhibits overactivation of macrophage inflammasome activity induced by Pg LPS - Jie Zhao, Wenjing Geng, Kefei Wan, Kailei Guo, Fengjun Xi, Xiangqun Xu, Xiujuan Xiong, Xu Huang,
Porphyromonas gingivalis lipopolysaccharide-induced macrophages modulate proliferation and invasion of head and neck cancer cell lines - ScienceDirect
Circular RNA PPP1CC promotes Porphyromonas gingivalis-lipopolysaccharide-induced pyroptosis of vascular smooth muscle cells by activating the HMGB1/TLR9/AIM2 pathway - Jie Liu, Yong Wang, Yaoyun Liao, Ying Zhou, Jijin Zhu, 2021
![InvivoGen] LPS-PG (Standard) : DAWINBIO](https://cdn.imweb.me/thumbnail/20201001/616646e7635ed.jpg "InvivoGen] LPS-PG (Standard) : DAWINBIO")
InvivoGen] LPS-PG (Standard) : DAWINBIO
Chlorogenic Acid as a Positive Regulator in LPS-PG-Induced Inflammation via TLR4/MyD88-Mediated NF-κB and PI3K/MAPK Signaling Cascades in Human Gingival Fibroblasts
Tetra- and Penta-Acylated Lipid A Structures of Porphyromonas gingivalis LPS Differentially Activate TLR4-Mediated NF-κB Signal Transduction Cascade and Immuno-Inflammatory Response in Human Gingival Fibroblasts | PLOS ONE
![Porphyromonas gingivalis lipopolysaccharide promotes T-hel per17 cell differentiation by upregulating Delta-like ligand 4 expression on CD14+ monocytes [PeerJ]](https://dfzljdn9uc3pi.cloudfront.net/2021/11094/1/fig-5-2x.jpg "Porphyromonas gingivalis lipopolysaccharide promotes T-hel per17 cell differentiation by upregulating Delta-like ligand 4 expression on CD14+ monocytes [PeerJ]")
Porphyromonas gingivalis lipopolysaccharide promotes T-hel per17 cell differentiation by upregulating Delta-like ligand 4 expression on CD14+ monocytes [PeerJ]
Anti-P.gingivalis mAb (PG antibody)（Monoclonal, AW1-4） | Antibody | MBL Life Sience -ASIA-
Hemin-Dependent Modulation of the Lipid A Structure of Porphyromonas gingivalis Lipopolysaccharide | Infection and Immunity
Porphyromonas gingivalis lipopolysaccharides act exclusively through TLR4 with a resilience between mouse and human | Scientific Reports
Porphyromonas Gingivalis in the Pathogenesis of Alzheimer's Disease and Its Therapeutic Target
Frontiers | Pathogenesis of Important Virulence Factors of Porphyromonas gingivalis via Toll-Like Receptors
